Preliminary experiments suggest conolidine may well inhibit certain ion channels, minimizing neuronal excitability and restricting ache indicators. This system is especially appropriate in neuropathic ache, in which irregular signaling will cause persistent soreness. In addition, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to soreness notion. Compu... https://johnu602dys1.blogdal.com/profile